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- Wu, X. C., et al.
(författare)
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CPSDv0: a forest stand structure database for plantation forests in China
- 2023
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Ingår i: Big Earth Data. - : Informa UK Limited. - 2096-4471 .- 2574-5417. ; 7:1, s. 212-230
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Tidskriftsartikel (refereegranskat)abstract
- Forest stand structure is not only a crucial factor for regulating forest functioning but also an important indicator for sustainable forest management and ecosystem services. Although there exists a few national/global structure databases for natural forests, a country-wide synthetic structure database for plantation forests over China, the world's largest player in plantation forests, has not been achieved. In this study, we built a country-wide synthetic stand structure database by surveying more than 600 peer-reviewed literature. The database covers tree species, mean stand age, mean tree height, stand density, canopy coverage, diameter at breast height, as well as the associated ancillary in-situ topographical and soil properties. A total of 594 published studies concerning diverse forest stand structure parameters were compiled for 46 tree species. This first synthesis for stand structure of plantation forests over China supports studies on the evolution/ health of plantation forests in response to rapid climate change and intensified disturbances, and benefits country-wide sustainable forest management, future afforestation or reforestation planning. Potential users include those studying forest community dynamics, regional tree growth, ecosystem stability, and health, as well as those working with conservation and sustainable management.
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| 2. |
- Zhang, X. L., et al.
(författare)
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Germinal matrix hemorrhage induces immune responses, brain injury, and motor impairment in neonatal rats
- 2023
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Ingår i: Journal of Cerebral Blood Flow and Metabolism. - : SAGE Publications. - 0271-678X .- 1559-7016. ; 43:2_suppl, s. 49-65
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Tidskriftsartikel (refereegranskat)abstract
- Germinal matrix hemorrhage (GMH) is a major complication of prematurity that causes secondary brain injury and is associated with long-term neurological disabilities. This study used a postnatal day 5 rat model of GMH to explore immune response, brain injury, and neurobehavioral changes after hemorrhagic injury. The results showed that CD45(high)/CD11b(+) immune cells increased in the brain after GMH and were accompanied by increased macrophage-related chemokine/cytokines and inflammatory mediators. Hematoma formed as early as 2 h after injection of collagenase VII and white matter injury appeared not only in the external capsule and hippocampus, but also in the thalamus. In addition, GMH caused abnormal motor function as revealed by gait analysis, and locomotor hyperactivity in the elevated plus maze, though no other obvious anxiety or recognition/memory function changes were noted when examined by the open field test and novel object recognition test. The animal model used here partially reproduces the GMH-induced brain injury and motor dysfunction seen in human neonates and therefore can be used as a valid tool in experimental studies for the development of effective therapeutic strategies for GMH-induced brain injury.
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